According to this model, hormonal changes within the body promote calorie deposition in adipose tissue, exacerbate hunger, and lower energy expenditure resulting in obesity.
The presence of insulin in the body is said to affect body weight because the hormone plays a major role in modulating the activities of several enzymes that are responsible for promoting uptake, retension, and storage of fat in adipose tissue.
In summary, the carbohydrate-insulin model of obesity puts forth that "diets with a high proportion of carbohydrate elevate insulin
secretion and thereby suppress the release of fatty acids from
adipose tissue into the circulation and direct circulating fat toward
adipose storage and away from oxidation by metabolically active
tissues such as heart, muscle and liver."
Relationship Between Food Intake and Weight
In carbohydrate-insulin model, the type of food intake i.e. carbohydrates is what results to weight gain as increased carbohydrates ingestion is believed to result in the elevated secretion of insulin, which in turn results in the convertion of blood sugar or glucose into fat for storage in the body's adipose tissue.
Consequently, in this model, the intake of foods that are high in carbohydrates result in weight gain as a result of the activities of insulin in the body.
Arguments against this model include the fact that high dietary fat can cause weight gain and obesity, usually at the expense of carbohydrates.
The model is also considered by many researchers to be too simple in how it explains obesity and according to nutrition experts, "despite the popularity of low-carbohydrate diets, there are no long-term conclusive data to suggest these diets may increase metabolic function, suppress appetite, or lead to sustained weight loss."
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